Three-Quarters of Recovered Coronavirus Patients Have Heart Damage Months Later, Study Finds

[eastcst84]

https://people.com/health/three-quarters-recovered-coronavirus-patients-have-heart-damage-months-later/?fbclid=IwAR2v_AEWz_uzEz3_XgOLoUDEZZdvpxAix_CGiEaZ0u2cXD5k07cI4w28rPU

 

Thoughts.

[Scott]

Off-topic post removed. 

 

[Jingthing]

It's an extremely threatening virus. It's not like a seasonal flu. 

[elliss]

Covid 19 .   Our World is changing .

  Five years hence , anyone like too predict the near future .

[geriatrickid]

It has been reported since Febuary that Autopsied patients show blood clots that have damaged vital organs such as the heart, brain, kidneys and liver.  This is why  hydroxychloroquine should not be  prescribed for patients: It  causes worse damage in  patients with heart disease and irreversibly damages the heart.

 

The most recent illustration; https://www.sciencealert.com/covid-19-patient-autopsies-show-blood-clots-in-almost-every-organ-pathologist-says

 

The idiots who compare this to the seasonal flu ignore the fact that the lung damage makes patients more susceptible to future  respiratory infections. The next infection will  further weaken the patient or result in death. Scarred hearts and kidneys do not repair themselves. Instead they deteriorate over time. 

 

[Sheryl]

Presumably this refers to recovered hospitalized patients not people who had mild disease or were asymptomatic.

 

It has become clear that severe COVID-19 is not just viral pneumonia as originally thought but rather a very complex multi-organ disease. With long term sequalae for survivors.

 

Starting to look like simple COVID and the severe form are really very different clinical entities, rather like Dengue vs Dengue Hemorrhagic Fever. Still a lot remains to be known about what causes severe COVID-19 disease, why only some people get it etc. This gap needs to close as soon as possible. It has potentially major implications for use of a vaccine, for example.

 

 

 

[asiacurious]

32 minutes ago, Sheryl said:

Presumably this refers to recovered hospitalized patients not people who had mild disease or were asymptomatic.

 

It has become clear that severe COVID-19 is not just viral pneumonia as originally thought but rather a very complex multi-organ disease. With long term sequalae for survivors.

 

Starting to look like simple COVID and the severe form are really very different clinical entities, rather like Dengue vs Dengue Hemorrhagic Fever. Still a lot remains to be known about what causes severe COVID-19 disease, why only some people get it etc. This gap needs to close as soon as possible. It has potentially major implications for use of a vaccine, for example.

 

 

The study in the OP's link is a German study of recovered patients, not asymptomatic cases.  BUT there is this out of the University of California San Francisco highlights the issue further, and it may not matter how severely one gets the virus.

 

Here are a few snippets from a rather long article (emphasis added)

 

https://www.ucsf.edu/magazine/covid-body?fbclid=IwAR3TzLPQIICW-B-BU0kyqmCBjhQf9u2qtVb1S-65SzxFRpv_dYL9gArZ7E8

 

Quote

The novel coronavirus, an RNA virus named SARS-CoV-2, has become notorious for its skill at breaking and entering human cells. Its tools of choice are the protein spikes protruding from its surface – a feature that distinguishes all coronaviruses. The spikes of SARS-CoV-2 are the crème de la crème: By the luck of the evolutionary draw, they are able to easily grab hold of protein gates on human cells known as ACE2 receptors and, like jackknives, pry these gates open.

 

[snip]

 

Since March, Marcus has co-led one of the largest community surveys to better understand the spread of SARS-CoV-2 and its myriad effects. The study, dubbed COVID-19 Citizen Science, has so far enrolled more than 27,000 people; anyone with a smartphone can participate. Marcus plans to also start collecting data from wearable devices, including Fitbits and Zio patches, which wirelessly monitor heart rhythms. “There may be large numbers of people who are suffering from cardiovascular effects of COVID-19 in the absence of other symptoms,” Marcus says. “I’m worried we’re missing those cases.”

 

It stands to reason that SARS-CoV-2 affects the heart. After all, heart cells are flush with ACE2 receptors, the virus’s vital port of entry. And, indeed, laboratory experiments suggest that the virus can enter and replicate in cultured human heart cells, says Bruce Conklin, MD, a professor of medicine and an expert in heart-disease genetics at UCSF and the Gladstone Institutes.